Question

sperrys initial version of the chemoaffinity hypothesis of axon growth has difficulty accounting for: the amount of neurons with connectivity issues in the auditory cortex the ability of axons to grow to their correct targets the time it took for the axon growth the occur the ability of axons to follow exactly the same circuitous route to their target in every member of a species the results of sperrys own eye - rotation regeneration experiments

Explanation:

Sperry's chemoaffinity hypothesis initially proposed that axons grow to specific targets due to chemical markers. However, the idea that axons follow exactly the same circuitous route in every member of a species is hard to reconcile with the hypothesis. The hypothesis focuses more on target - seeking based on chemical affinity rather than the exact, same circuitous path for all individuals. The amount of neurons with connectivity issues in the auditory cortex is not a main focus of the initial hypothesis challenge. The ability of axons to grow to correct targets is what the hypothesis was about (so it doesn't have difficulty accounting for that). The time of axon growth is not a key problem area for the initial chemoaffinity hypothesis. And Sperry's own eye - rotation regeneration experiments actually supported aspects of his hypothesis.

Answer:

the ability of axons to follow exactly the same circuitous route to their target in every member of a species